Abstract
The neutrophil is important in host defense against bacterial and fungal infections (1). That the neutrophil is capable of serving in this crucial role is made possible by-virtue of its unique ability to induce microbial killing, in part, by exposing ingested microorganisms to toxic oxygen metabolites, including superoxide anion ((math)) — products of the respiratory burst, which commences during phagocytosis (2).
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Forehand, J.R., Bomalski, J.S., Johnston, R.B. (1991). Mechanisms of Lipopolysaccharide Priming for Enhanced Respiratory Burst Activity in Human Neutrophils. In: Hörl, W.H., Schollmeyer, P.J. (eds) New Aspects of Human Polymorphonuclear Leukocytes. Advances in Experimental Medicine and Biology, vol 297. Springer, Boston, MA. https://doi.org/10.1007/978-1-4899-3629-5_6
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DOI: https://doi.org/10.1007/978-1-4899-3629-5_6
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