Abstract
Why should a book such as a book on the Physiology and Pathophysiology of the Islets of Langerhans be published, bought, and even read?
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References
Langerhans, P (1869) Beiträge zur mikroskopischen. Anatomie der Beauchspercheldrüse (Thesis). Friedrich-Wilhelms-Universität, Berlin.
Froguel P, Zouali H, Vionnet N, Velho G, Vaxillaire M, Sun F, Lesague S, Stoffel M, Takeda J, Passa P, Permuti A, Beckamn J, Bell GI, Cohen D (1993) Familial hyperglycaemia due to mutations in glucokinase. N. Eng. J. Med. 328: 697–702.
Porte Jr, D (1991) β-cells in type II diabetes mellitus. Diabetes 40: 166–180.
Soria B, Andreu E, Pertusa JG, Martin F, Ripoll C, Sanchez-André JV (1996) Target molecules as candidates to be defective in stimulus-secretion coupling in type II diabetes. Endocrinologia 43(9): 321–328.
Andreu E, Soria B, Sanchez-Andrés JV (1997) Oscillations of the gap junction electrical coupling in the mouse pancreatic islet of Langerhans. J. Physiol. (Lond) 498(3): 753–761.
Ashcroft FM, Rorsman P (1991) Electrophysiology of the pancreatic β-cell. Progress in Biophysics and Molecular Biology 54: 87–147.
Valdeolmillos M, Nadal A, Contreras D, Soria B (1992) The relationship between glucose-induced K(ATP) channel closure and the rise in [Ca2+]i in single mouse pancreatic B-cells. J. Physiol. (Lond) 455: 173–186.
Gembal M, Detimary P, Gilon P, Gao ZY, Henquin JC (1993) Mechanisms by which glucose can control insulin release independently from its action on adenosine triphosphate-sensitive K+ channels in mouse B cells. J. Clin. Invest. 91: 871–880.
Cook DL, Hales CN (1984) Intracellular ATP directly blocks K+ channels in pancreatic β-cells. Nature 311: 271–273.
Ashcroft FM, Harrison DE, Ashcroft SJH (1984) Glucose induces closure of single potassium channels in isolated rat pancreatic β-cells. Nature 312: 446–448.
Atwater I, Rojas E, Soria B (1986) Biophysics of the Pancreatic β-cell. Plenum Press, New York.
Ashford MLJ, Sturgess NC., Cook DL, Hales CN (1986) K-channels in an Insulin-secreting Cell Line. In: Biophysics of the Pancreatic β-cell (Atwater, Rojas, and Soria, eds) Plenum Press, New York pp. 69–76.
Aguilar-Bryan L, Nichols CG, Wechsler SW, Clement JP, Boyd III AE, Gonzalez G, Herrera-Sosa H, Nguyk K, Bryan J, Nelson DA (1995) Cloning of the beta-cell high-affinity sulphonylurea receptor: a regulator of insulin secretion. Science 268: 372–373.
Inagaki N, Gonoi T, Clement JP, Namba N, Inazawa J, Gonzalez G, Aguilar-Bryan L, Seino S, Bryan J (1995) Reconstitution of KATP: an inward rectifier subunit plus the sulphonylurea receptor. Science 270: 1166–1170.
Kane C., Shepperd RM, Squires PE, Johnson PRV, James RFL, Milla PJ, Aynsley-Green A, Lindley KJ, Dunne MJ (1996) Loss of functional KATP channels in pancreatic β-cells causes persistent hyperinsulinaemic hypoglyccemia of infancy. Nature Medicine 2(12): 1344–1347.
Jacobson G, Bean AJ, Scheller RH, Juntti-Berggren L, Deeney JT, Berggren P-O (1994) Identification of synaptic proteins and their isoform mRNAs in compartiments of pancreatic endocrine cells. Proc. Natl. Acad. Sci. USA 91: 12487–12491.
Martin F, Moya F, Gutierrez LM, Reig JA, Soria B (1995) Role of syntaxin in mouse pancreatic β-cell. Diabetologia 38: 860–863.
Martin F, Salinas E, Vazquez J, Soria B, Reig JA (1996) Inhibition of insulin release by synthetic peptides shows that the H3 region at the C-terminal domain of syntaxin-1 is crucial for calcium but not for GTP-S-induced secretion. Biochem. J. 320: 201–205.
Valdeolmillos M, Santos R, Contreras D, Soria B, Rosario LM (1989) Glucose-induced oscillations of intracellular Ca2+ concentration resembling bursting electrical activity in single mouse islets of Langerhans. FEBS Letters 259: 19–23.
Santos RM, Rosario LM, Nadal A, GarcÃa-Sancho J, Soria B, Valdeolmillos M (1991) Widespread synchronous [Ca2+]i oscillations due to bursting electrical activity in single pancreatic islets. Pflügers Archiv 418: 417–422.
Martin F, Sanchez-Andrés JV, Soria B (1995) Slow [Ca2+] oscillations induced by ketoisocaproate in mouse pancreatic islets. Diabetes 44: 300–305.
Martin F, Soria B (1995) Amino acid induced [Ca2+]i oscillations in single mouse pacreatic islets. J. Physiol. (Lond) 486: 361–371.
Valdeolmillos M, Nadal A, Soria B, GarcÃa-Sancho J (1993) Fluorescence digital image analysis of glucose-induced [Ca2+]i oscillations in mouse pancreatic islet of Langerhans. Diabetes 42: 1210–1214.
Soria B, Chanson M, Giordano E, Bosco D, Meda P (1991) Ionic channels in glucose responsive and unresponsive B-cells. Diabetes 40: 1069–1078.
Soria B (1994) First Meeting of the Pancreatic Islet Study Group. Diabetologia 7: 27–30.
Malaisse WJ (1995) Non-insulin dependent diabetes mellitus and islet B-cell mitochondrial glycerophosphate dehydrogenase deficiency. Diabetes Med. 12: 479–481.
Boyd III AE, Hill RS, Nelson TY, Oberwetter JM, Berg M (1986) In: Biophysics of the Pancreatic β-cell (I. Atwater, E. Rojas and B. Soria, eds). Plenum Press, New York, pp. 479–481.
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Soria, B. (1997). Why are Islets so Important?. In: Soria, B. (eds) Physiology and Pathophysiology of the Islets of Langerhans. Advances in Experimental Medicine and Biology, vol 426. Springer, Boston, MA. https://doi.org/10.1007/978-1-4899-1819-2_1
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DOI: https://doi.org/10.1007/978-1-4899-1819-2_1
Publisher Name: Springer, Boston, MA
Print ISBN: 978-1-4899-1821-5
Online ISBN: 978-1-4899-1819-2
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