Abstract
Disturbances of calcium metabolism are well documented in renal calcium-stone disease and hypercalciuria is the most commonly-occurring metabolic deviation (1). Idiopathic hypercalciuria is considered to be caused by an increased gastrointestinal absorption of calcium, whereas a primary renal leak of calcium with the development of secondary hyperparathyroidism (HPT) is unusual, as judged by present diagnostic tools.
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© 1989 Springer Science+Business Media New York
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Fellström, B. et al. (1989). The Possibility of a Specific Calcium Sensor/Receptor Function of Proximal Renal Tubular Cells. In: Walker, V.R., Sutton, R.A.L., Cameron, E.C.B., Pak, C.Y.C., Robertson, W.G. (eds) Urolithiasis. Springer, Boston, MA. https://doi.org/10.1007/978-1-4899-0873-5_3
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DOI: https://doi.org/10.1007/978-1-4899-0873-5_3
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