The Possibility of a Specific Calcium Sensor/Receptor Function of Proximal Renal Tubular Cells
Disturbances of calcium metabolism are well documented in renal calcium-stone disease and hypercalciuria is the most commonly-occurring metabolic deviation (1). Idiopathic hypercalciuria is considered to be caused by an increased gastrointestinal absorption of calcium, whereas a primary renal leak of calcium with the development of secondary hyperparathyroidism (HPT) is unusual, as judged by present diagnostic tools.
KeywordsProximal Tubule Tubular Cell Parathyroid Adenoma Extracellular Calcium Proximal Tubular Cell
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