Urinary Calcium After Oral Glucose Ingestion in Calcium Stone Formers: A Simple Provocative Test for Predicting Stone Recurrence
Although the various metabolic abnormalities underlying Ca stone disease have been extensively studied, none of these except abnormalities in 24-h urine samples appear to be of any practical significance as predictors of stone risk (1–3). This is an issue of practical relevance. Previous studies have shown that urinary Ca excretion rises after glucose ingestion (4, 5), a fact that could account for hypercalciuria in some Ca stone formers who are overweight. In the present study, we examined increases in urinary Ca excretion following glucose ingestion in order to determine if there was some relation between these incidents in the evolution of Ca stone disease. This was a retrospective study and could, therefore, constitute a reliable predictive index for Ca stone recurrence.
KeywordsGlucose Ingestion Stone Former Stone Disease Stone Risk Absorptive Hypercalciuria
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AL Strauss, FL Coe, L Deutsch, and JH Parks, Am. J. Med.
72: 17 (1982).PubMedCrossRefGoogle Scholar
CYC Pak, J. Urol.
127: 855 (1982).PubMedGoogle Scholar
E Laerum, Scand. J. Urol. Nephrol.
18: 67 (1984).PubMedCrossRefGoogle Scholar
RA De Fronzo, CR Cooke, R Andres, and PJ David, J. Clin. Invest.
55: 845 (1975).CrossRefGoogle Scholar
G Boner, M Newton, and RE Rieselbach, Kidney Int.
3: 24 (1973).PubMedCrossRefGoogle Scholar
CYC Pak, M Ohata, EC Lawrence, and W Snyder, J. Clin. Invest.
54: 387 (1974).PubMedCrossRefGoogle Scholar
L Lemann, WF Piering, and EJ Lennon, NEJM
280: 232 (1969).PubMedCrossRefGoogle Scholar
FP Muldowney, R Treang, and MF Moloney, Kidney Int.
22: 292 (1982).PubMedCrossRefGoogle Scholar
DA McCarron, LI Rankin, WM Bennett, S Krutzik, MR McLung, and FC Luft, Am. J. Nephrol.
1: 84 (1981).PubMedCrossRefGoogle Scholar
DV Weber, FL Coe, JH Parks, MSL Dunn, and V Tembe, Annals Int. Med.
90: 180 (1979).CrossRefGoogle Scholar
P Jaeger, L Portmann, JM Ginalski, AF Jacquet, E Tember, and P Burckhardt, Kidney Int.
29: 563 (1986).PubMedCrossRefGoogle Scholar
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