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Urolithiasis pp 355-358 | Cite as

Calcitonin and Parathyroid Hormone Provocative Tests in Fasting Hypercalciuria

  • P. Messa
  • G. Mioni
  • D. Montanaro
  • M. Messa
  • M. Adorati
  • S. Adami
  • M. Costantini
  • G. C. Englaro

Abstract

A primary role for bone as the source of Ca in fasting hypercalciuria (FH) of stone formers (SF) has been suggested by abundant evidence (1–3), Seventeen years ago, Libermann and de Vries (4) proposed a pathogenetic model for the explanation of the primary hypercalciuric states in which some, as yet undefined, deregulation of calcitonin and/or parathyroid hormone (PTH) secretion was hypothesized. Since then, many contradictory studies have been performed in regard to PTH status in hypercalciuric SF. However, little, if any, further information has been added on the role of calcitonin in this metabolic disorder. The aim of the present study was to investigate this matter.

Keywords

Stone Former Calcitonin Level Idiopathic Hypercalciuria Serum Calcitonin Phosphate Load 
These keywords were added by machine and not by the authors. This process is experimental and the keywords may be updated as the learning algorithm improves.

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Copyright information

© Springer Science+Business Media New York 1989

Authors and Affiliations

  • P. Messa
    • 1
  • G. Mioni
    • 1
  • D. Montanaro
    • 1
  • M. Messa
    • 1
  • M. Adorati
    • 1
  • S. Adami
    • 3
  • M. Costantini
    • 2
  • G. C. Englaro
    • 2
  1. 1.Institute of NephrologyVeronaItaly
  2. 2.Institute of Nuclear Medicine-UdineVeronaItaly
  3. 3.University of VeronaVeronaItaly

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