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Calcium Transport by Red Cells from Hypercalciuric Stone Formers

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Urolithiasis
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Abstract

It is commonly recognized that hypercalciuria is a risk factor for stone formation (1). Hyperabsorption of intestinal calcium (2), a renal leak of calcium, and/or increased bone resorption (3, 4) have been implicated as mechanisms of hypercalciuria.

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References

  1. CYC Pak, C Fetner, et al., Evaluation of calcium urolithiasis in ambulatory patients, Am. J. Med. 64: 979 (1978).

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  2. PH Henneman, PH Benedict, et al., Idiopathic hypercalciuria, NEJM 259: 802 (1958).

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  3. CYC Pak, Physiological basis for absorptive and renal hypercalciuria, Am. J. Physiol. 237: F415 (1979).

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  5. RW Gray, DR Wilz, et al., The importance of phosphate in regulating plasma 1,25(OH)2D levels in humans; studies in healthy subjects in calcium stone formers and in patients with primary hyperparathyroidism, J. Clin. Endocrinol. Metab. 259: 802 (1979).

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  6. PG Branan, S Morawski, et al., Idiopathic hypercalciuria, NEJM 259: 802 (1979).

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  7. B Pinto, MA Vilanova, et al., Kinetic differences of the calcium binding protein in absorptive hypercalciuric renal stone formers, Urol. Res. 13: 277 (1985).

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© 1989 Springer Science+Business Media New York

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Pinto, B., Pujol, A. (1989). Calcium Transport by Red Cells from Hypercalciuric Stone Formers. In: Walker, V.R., Sutton, R.A.L., Cameron, E.C.B., Pak, C.Y.C., Robertson, W.G. (eds) Urolithiasis. Springer, Boston, MA. https://doi.org/10.1007/978-1-4899-0873-5_108

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  • DOI: https://doi.org/10.1007/978-1-4899-0873-5_108

  • Publisher Name: Springer, Boston, MA

  • Print ISBN: 978-1-4899-0875-9

  • Online ISBN: 978-1-4899-0873-5

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