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Abstract

Cerebral venous thrombosis is associated with a variety of systemic illnesses, sepsis, pregnancy, coagulation defects, and autoimmune states. Deficiencies of antithrombin III, protein C, protein S, and factor V Leiden, or an increase in anticardiolipin A antibodies may be identified in patients with this disorder, but a precise pathogenesis may not be identifiable in 25% to 35% of patients.1 The disease is frequently overlooked clinically and on CT imaging, in part due to a low clinical suspicion of the disease stemming from its ability to simulate the clinical appearance of benign intracranial hypertension, neoplasms, encephalitis, or cerebrovascular accident.2 However, MRI with contrast, MR venography, and CT angiography all enhance reader sensitivity to this disease. The prognosis of the disease is unpredictable and variable. Mortality rates up to 33% can be reported depending on the underlying disease state and the presence or absence of hemorrhagic venous infarcts.3

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© 2002 Springer Science+Business Media New York

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Morris, P. (2002). Venous Thrombotic Disease. In: Interventional and Endovascular Therapy of the Nervous System. Springer, New York, NY. https://doi.org/10.1007/978-1-4757-3673-1_14

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  • DOI: https://doi.org/10.1007/978-1-4757-3673-1_14

  • Publisher Name: Springer, New York, NY

  • Print ISBN: 978-1-4419-2903-7

  • Online ISBN: 978-1-4757-3673-1

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