Gout encompasses a spectrum of diseases caused by precipitation of uric acid crystals in tissue. The gouty disorders include (1) acute monarticular arthritis caused by uric acid crystals in joints; (2) nephrolithiasis; (3) soft tissue deposits of urate crystals known as tophi; and (4) uric acid renal disease. Gout is about six times more common in men than in women,1 although there is evidence that the relative prevalence of gout in women has increased.2 The prevalence of gout increases with age, and it is more common in persons of African or Polynesian ancestry.
KeywordsUric Acid Serum Uric Acid Serum Uric Acid Level Triamcinolone Acetonide Acute Gout
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- 6.Joseph J, McGrath H. Gout or ‘pseudogout’: how to differentiate crystal-induced arthropathies. Geriatrics 1995; 50(4):13–39.Google Scholar
- 7.Beutler A, Schumacher HR. Gout and ‘pseudogout’: when are arthritic symptoms caused by crystal deposition? Postgrad Med 1994;2:103–20.Google Scholar
- 8.Alloway JA, Moriarty MJ, Hoogland YT, et al. Comparison of triamcinolone acetonide with indomethacin in the treatment of acute gouty arthritis. J Rheumat 1993;20:1383–5.Google Scholar
- 10.Corkill MM. Gout. NZ Med J 1994;107:337–9.Google Scholar