Vitreous Changes and Macular Disease

  • A. E. Jalkh
  • C. L. Trempe


Strong adhesions exist normally between the vitreous cortex and the retina in the macula (1). They may play a role in the occurrence and prognosis of macular diseases, particularly macular breaks, preretinal macular fibrosis (idiopathic or secondary), and cystoid macular edema. These conditions are directly related to vitreous changes, especially those producing vitreous traction. Macular conditions unrelated to vitreous changes such as senile macular degeneration and hereditary macular degenerations are not discussed here. Macular changes that result from peripheral uveitis are described in Chapter 7.


Macular Hole Cystoid Macular Edema Posterior Vitreous Detachment Macular Disease Idiopathic Macular Hole 
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  1. (1).
    GRIGNOLO, A. — Fibrous components of the vitreous body. Arch. Ophthalmol., 1952, 47, 760–774.CrossRefGoogle Scholar
  2. (2).
    MAUMENEE, A.E. — Further advances in the study of the macula. Arch. Ophthalmol., 1967, 78, 151–165.PubMedCrossRefGoogle Scholar
  3. (3).
    AABERG, T. M. — Macular holes: a review. Surv. Ophthalmol., 1970, 15, 139–162.Google Scholar
  4. (4).
    SCHEPENS, C.L. — Fundus changes caused by alterations of the vitreous body. Am. J. Ophthalmol., 1955, 39, 631–633.PubMedGoogle Scholar
  5. (5).
    AVILA, M.P., JALKH, A.E., MURAKAMI, K., TREMPE, C.L., SCHEPENS, C. L. — Biomicroscopic study of the vitreous in macular breaks. Ophthalmology, 1983, 90, 1277–1283.PubMedGoogle Scholar
  6. (6).
    TOLENTION, F.I., SCHEPENS, C.L., FREEMAN, H.M. — Vitreoretinal disorders: diagnosis and management. Philadelphia: W.B. Saunders, 1976, 149, 400–410.Google Scholar
  7. (7).
    TREMPE, C.L., WEITER, J.J., FURAKAWA, H. — Fellow eyes in cases of macular hole: biomicroscopic study of the vitreous. Arch. Ophthalmol., 1986, 104, 93–95.PubMedCrossRefGoogle Scholar
  8. (8).
    Bronstein, M.A., Trempe, C.L., Freeman, H.M. — Fellow eyes of eyes with macular holes. Am. J. Ophthalmol., 1981, 92, 757–761.PubMedGoogle Scholar
  9. (9).
    WISE, G.N. — Clinical features of idiopathic preretinal macular fibrosis. Schoenberg lecture. Am. J. Ophthalmol., 1975, 79, 349–357.PubMedGoogle Scholar
  10. (10).
    SIDD, R.J., FINE, S.L., OWENS, S.L., PATZ, A. — Idiopathic preretinal gliosis. Am. J. Ophthalmol., 1982, 94, 44–48.PubMedGoogle Scholar
  11. (11).
    LAQUA, H., MACHEMER, R. — Glial cell proliferation in retinal detachment (massive periretinal proliferation). Am. J. Ophthalmol, 1975, 80, 602–618.PubMedGoogle Scholar
  12. (12).
    WISE, G.N. — Relationship of idiopathic preretinal macular fibrosis to posterior vitreous detachment. Am. J. Ophthalmol., 1975, 79, 358–362.PubMedGoogle Scholar
  13. (13).
    HIROKAWA, H., JALKH, A.E., TAKAHASHI, M., TAKAHASHI, M., TREMPE, C. L., SCHEPENS, C.L. — Role of the vitreous in idiopathic preretinal macular fibrosis. Am. J. Ophthalmol., 1986, 101, 166–169.PubMedGoogle Scholar
  14. (14).
    SCHEPENS, C. L., AVILA, M. P., JALKH, A. E., TREMPE, C.L. — Role of the vitreous in cystoid macular edema. Surv. Ophthalmol., 1984, 28 (5S), 499–504.PubMedCrossRefGoogle Scholar

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© Springer Science+Business Media New York 1987

Authors and Affiliations

  • A. E. Jalkh
  • C. L. Trempe

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