Abstract
Moderator (Oscar Sudilovsky): After such interesting presentations I think it is time to concentrate on the mechanistic similarities between tumor promotion and progression. Dr. Pitot, if I interpret him correctly, said that the effect of promoting agents can extend to cells in the stage of progression, while Dr. Slaga expressed the opinion that both processes exist as a continuum. So I would like to start the discussion by asking Dr. Pitot how he would contrast his belief with those of Dr. Slaga.
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References
O. Sudilovsky and T. K. Hei: Aneuploid nuclear DNA content in some enzyme-cultured rat liver foci during tumor promotion. Fed. Proc. 42:7 (1983).
J. H. Wang, L. Hinrichsen, C. Mansilla and O. Sudilovsky: Hepatocarcinogenesis: Aneuploid nuclear DNA content in enzyme-altered foci (EAF) in rats. Lab. Invest. 58:100A (1988).
S. L. Robbins and R. S. Cotran, “The Pathologic Basis of Disease”, 2nd Edition, W. B. Saunders Company, Philadelphia, PA, 1979, p. 182.
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© 1991 Plenum Press, New York
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Sudilovsky, O., Pitot, H.C., Liotta, L.A. (1991). Discussion of Session. In: Sudilovsky, O., Pitot, H.C., Liotta, L.A. (eds) Boundaries between Promotion and Progression during Carcinogenesis. Basic Life Sciences, vol 57. Springer, Boston, MA. https://doi.org/10.1007/978-1-4684-5994-4_6
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DOI: https://doi.org/10.1007/978-1-4684-5994-4_6
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