Abstract
It is well-established that ischemia leads to very complex and heterogenous changes in microcirculation and metabolism of the brain /Siesjö, 1981; Raichle, 1983; Welsh, 1984/. Although the severity of brain damage depends mainly on the degree and duration of the decrease of cerebral blood flow /CBF/, and hence the depletion of macroerg phosphates, mitochondria seem quite resistant to ischemia /Ikrényi et al., 1976; Ginsberg et al., 1977; Rehncrona et al., 1979; Siesjö, 1981; Dóra, 1985b/.
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© 1986 Plenum Press, New York
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Dóra, E., Greenberg, J.H., Tanaka, K., Gonatas, N.H., Reivich, M. (1986). Microcirculation and Mitochondrial Function in Focal Brain Ischemia. In: Longmuir, I.S. (eds) Oxygen Transport to Tissue VIII. Advances in Experimental Medicine and Biology, vol 200. Springer, Boston, MA. https://doi.org/10.1007/978-1-4684-5188-7_26
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DOI: https://doi.org/10.1007/978-1-4684-5188-7_26
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