Abstract
The experiments reported in this paper were designed to test one of several possible mechanisms that may underly the phenomenon of metabolic cooperation between normal and HG-PRT deficient cells, namely transfer of some intermediate of the purine metabolism from cell to cell.
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References
H. Subak-Sharpe, R.R. Bark and J.D. Pitts (1966): nMetabolic cooperation by cell to cell transfer between genetically different mammalian cells in tissue culture. Heredity 21, 342–343.
H. Subak-Sharpe, R.R. Bark and J.D. Pitts (1969): Metabolic cooperation between biochemically marked mammalian cells in tissue culture. J. Cell Sci. 4, 353–367.
C.H.M.M. de Bruyn and T.L. Oei (1973): Purine metabolism in intact erythrocytes from controls and HG-PRT deficient individuals. These Proceedings.
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© 1974 Plenum Press, New York
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Oei, T.L., de Bruyn, C.H.M.M. (1974). Studies on Metabolic Cooperation Using Different Types of Normal and Hypoxanthine-Guanine Phosphoribosyltransferase (HG-PRT) Deficient Cells. In: Sperling, O., De Vries, A., Wyngaarden, J.B. (eds) Purine Metabolism in Man. Advances in Experimental Medicine and Biology, vol 41A. Springer, Boston, MA. https://doi.org/10.1007/978-1-4684-3294-7_29
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DOI: https://doi.org/10.1007/978-1-4684-3294-7_29
Publisher Name: Springer, Boston, MA
Print ISBN: 978-1-4684-3296-1
Online ISBN: 978-1-4684-3294-7
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