Abstract
The main objective of experimental atherosclerosis is to start in anima vili the atherogenic process in such a way as to be able to study its cause or causes, its development and its effects. However, such an ambitious program is nearly impossible to carry out if one wishes to extrapolate the experimental observations in spontaneous human atherosclerosis. Indeed, the latter is a multifactorial phenomenon in which metabolic elements play without a doubt a major but certainly not the only role.
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Vastesaeger, M., Peeters, H., Blaton, V., Petrovas, C., Mortelmans, J. (1975). Some Aspects of the Chimpanzee as a Model for Experimental Atherosclerosis. In: Kritchevsky, D., Paoletti, R., Holmes, W.L. (eds) Lipids, Lipoproteins, and Drugs. Advances in Experimental Medicine and Biology, vol 63. Springer, Boston, MA. https://doi.org/10.1007/978-1-4684-3258-9_26
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DOI: https://doi.org/10.1007/978-1-4684-3258-9_26
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