Abstract
The local O2 supply of liver tissue during the development of hemorrhagic shock was investigated with O2 electrodes. It was found that local anoxia and disturbances in the microcirculation could be detected even in the early phase of hemorrhagic shock, when systolic blood pressure was still at or above 100 mm Hg. Fig 1 shows a typical example of early tissue anoxia in dog liver.
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References
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Kessler, M., Lang, H., Starlinger, H., Höper, J., Thermann, M. (1973). Aerobic and Anaerobic Glycolysis in the Liver after Hemorrhage. In: Kovách, A.G.B., Stoner, H.B., Spitzer, J.J. (eds) Neurohumoral and Metabolic Aspects of Injury. Advances in Experimental Medicine and Biology, vol 33. Springer, Boston, MA. https://doi.org/10.1007/978-1-4684-3228-2_29
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DOI: https://doi.org/10.1007/978-1-4684-3228-2_29
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