Abstract
The liberation of deposit histamine from mast cells and the role of histamine in immunological (anaphylactic) shock reactions is well established. Concerning endotoxin shock and various other forms of shock, however, the significance of histamine is still subject to discussion, although the histamine liberating effect of endotoxins from mast cells has been proved by Hinshaw et al. (1960).
Supported by Deutsche Forschungsgemeinschaft and Volkswagen-Stiftung
Access this chapter
Tax calculation will be finalised at checkout
Purchases are for personal use only
Preview
Unable to display preview. Download preview PDF.
References
Aüres D., and W.G. Clark: A Rotating Diffusion Chamber for C14O2. Determination as Applied to Inhibitor Studies on Mouse Mast Cell Tumor Histidine Decarboxylase. Analytical Biochem. 9 35 (1964).
Brocklehurst W.E. and S.C. Lahiri: J. Physiol. 160, 15 (1962).
Di Stefano H.S., A.D. Bass, H.F. Diermeier and J. Teppermann: Nucleic acid patterns in rat liver following hypophysectomy and growth hormone administration. Endocrinology 51 386, 1952.
Hinshaw L.B., J.A. Vick, C.H. Carlson and Y.L. Fan: Role of histamine in endotoxin shock. Proc. Soc. exp. Biol. Med. 104, 379 (1960).
Lillehei R.C., J.K. Longerbeam, J.H. Bloch and W.G. Manax: Hemo dynamic changes in endotoxin shock. In: Shock and Hypotension, The XII. Hahnemann Symposium. Ed. L.C. Millis and J.H. Moye Grune and Stretton, New York-London, P. 442, 1965.
Schauer A., I. Menzinger and L. Gielow: Increased histidine decarboxylase activity of rat lung in endotoxin shock. Nature (Lond.) 212, 1249, (1966).
Schauer A., L. Gielow: Beeinflussung der “induzierbaren” Histidindecarboxylase durch Gluco-corticoide nach Endotoxingabe. Naturwissenschaften, 54, 589/90 (1967).
Schauer, A., und L. Gielow: Steigerung der Histidindecarboxy-laseaktivität während des Endo-toxinschocks. Verh. Dt. Gesch. Path. 51, 271 (1967).
Schauer A., L. Gielow und R. Calvoer: Zur Pathogenese des Endotoxin-schocks. Klin. Wo. 65, 593 (1967).
Schauer A., L. Gielow und G. Geissl: Das Verhalten der Histidindecarboxylase und der Plasmakinine in Endo-toxinschock. Ref. Peptidsymp. anläßlich des 65. Geburtstags von Prof. Werle.
Schayer R.W., Z. Rothschild and P. Bizony: Increase in histidine decarboxylase activity of rat skin following treatment with compound 48/80. Am. J. Physiol. 196, 295 (1959).
Schayer R.W. and O.H. Ganley: Adaptive increase in mammalian histidine decarboxylase activity in response to nonspecific stress. Am. J. Physiol. 197, 721 (1959).
Schayer R.W.: Evidence that induced histamine is an intrinsic regulator of the micro-circulatory system. Am. J. Physiol. 202, 66 (1962).
Zweifach B.W., A.L. Nagler and L. Thomas: The role of epinephrine in the reactions produced by the endotoxins of gram negative bacteria II. The changes produced by endotoxin in the vascular reactivity to epinephrine in the rat mesoappendix and the isolated perfused rabbit ear. J. exp. Med. 104, 881 (1956).
Author information
Authors and Affiliations
Editor information
Editors and Affiliations
Rights and permissions
Copyright information
© 1970 Plenum Press, New York
About this chapter
Cite this chapter
Schauer, A., Gielow, L., Geissl, G. (1970). Behaviour of Histidine Decarboxylase (HDC) in Various Forms of Shock. In: Bertelli, A., Back, N. (eds) Shock: Biochemical, Pharmacological, and Clinical Aspects. Advances in Experimental Medicine and Biology, vol 9. Springer, Boston, MA. https://doi.org/10.1007/978-1-4684-3201-5_4
Download citation
DOI: https://doi.org/10.1007/978-1-4684-3201-5_4
Publisher Name: Springer, Boston, MA
Print ISBN: 978-1-4684-3203-9
Online ISBN: 978-1-4684-3201-5
eBook Packages: Springer Book Archive