Abstract
Some years ago this group suggested that microvascular endothelium may be considered as one of the regulators of kinin metabolism on the basis of the following data:
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a)
Arterial kininogen is slightly higher than that of venous (15).
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b)
Venous recovery of intraarterial bradykinin is very poor, indicating a high capillary clearance (1, 2).
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c)
During the reactive hyperemia following circulatory arrest, kininogen decreases (15).
With the aid of the National Research Council of Rome.
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Bavazzano, A., Sidell, N., Michelacci, S., Sicuteri, F. (1970). Local Ischemia as a Kininogen Depressant: Effect of Cortisone, Taurine and a Kallikrein Inhibitor. In: Sicuteri, F., e Silva, M.R., Back, N. (eds) Bradykinin and Related Kinins. Advances in Experimental Medicine and Biology, vol 8. Springer, Boston, MA. https://doi.org/10.1007/978-1-4684-3198-8_45
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