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“Lipolipin”: A Glycoprotein Inhibitor of Postheparin Plasma Lipoprotein Lipase

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Heparin

Part of the book series: Advances in Experimental Medicine and Biology ((AEMB,volume 52))

Abstract

It has been established that the uptake of circulating blood triglyceride fatty acids by extrahepatic tissues is mediated through the hydrolysis of triglycerides by lipoprotein lipase (LPL). LPL hydrolyzes triglycerides only in the presence of lipoproteins, and its activity is considered essential for a normal rate of clearance of triglycerides from plasma (15).

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Author information

Authors and Affiliations

  1. Connective Tissue Laboratory, Veterans Administration Hospital, Little Rock, Arkansas, 72206, USA

    Premanand V. Wagh

  2. Department of Biochemistry, University of Arkansas Medical Center, Little Rock, Arkansas, 72201, USA

    Premanand V. Wagh

Authors
  1. Premanand V. Wagh
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Editor information

Editors and Affiliations

  1. Department of Biological Chemistry, Washington University School of Medicine, St. Louis, Missouri, USA

    Ralph A. Bradshaw

  2. Department of Medicine, The Jewish Hospital, St. Louis, Missouri, USA

    Stanford Wessler

  3. Washington University School of Medicine, St. Louis, Missouri, USA

    Stanford Wessler

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© 1975 Plenum Press, New York

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Wagh, P.V. (1975). “Lipolipin”: A Glycoprotein Inhibitor of Postheparin Plasma Lipoprotein Lipase. In: Bradshaw, R.A., Wessler, S. (eds) Heparin. Advances in Experimental Medicine and Biology, vol 52. Springer, Boston, MA. https://doi.org/10.1007/978-1-4684-0946-8_25

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  • DOI: https://doi.org/10.1007/978-1-4684-0946-8_25

  • Publisher Name: Springer, Boston, MA

  • Print ISBN: 978-1-4684-0948-2

  • Online ISBN: 978-1-4684-0946-8

  • eBook Packages: Springer Book Archive

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