Abstract
In brain and other tissues, there has been a recent re-emphasis on the role of mitochondria in the cascade of events producing cell injury and death following anoxia/ischemia (e.g., Abe et al., 1995; Ankarcrona et al., 1995; Saris and Eriksson, 1995; Kluck et al., 1997; Yang et al., 1997).
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Pérez-Pinzón, M.A., Sick, T.J., Rosenthal, M. (1999). Mechanism(s) of Mitochondrial Hyperoxidation after Global Cerebral Ischemia. In: Eke, A., Delpy, D.T. (eds) Oxygen Transport to Tissue XXI. Advances in Experimental Medicine and Biology, vol 471. Springer, Boston, MA. https://doi.org/10.1007/978-1-4615-4717-4_21
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DOI: https://doi.org/10.1007/978-1-4615-4717-4_21
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