Abstract
Medication of subgroups of idiopathic calcium urolithiasis often aims at reducing an alkaline or insufficiently acidic urinary pH. Since (exogenous) acid loading is effective but instantly attacks the body’s buffer reservoirs, we tested the hypothesis that inhibition of postprandial gastric proton release into the gastric lumen may lead to proton diversion to kidney via blood, and to subsequent excretion via urine.
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GA Williams, RS Longley, EN Bowser et al, Parathyroid hormone secretion in normal man and in primary hyperparathyroidism: Role of histamine H2 receptors. J Clin Endocrinol Metab 52: 122 (1981).
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© 1994 Springer Science+Business Media New York
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Herrmann, U., Schwille, P.O., Manoharan, M., Gruber, H., Wenig, A. (1994). Oral Cimetidinein Humans - Evidence For Urine Acidification and Inhibition of Calcium Oxalate Crystallization. In: Ryall, R., Bais, R., Marshall, V.R., Rofe, A.M., Smith, L.H., Walker, V.R. (eds) Urolithiasis 2. Springer, Boston, MA. https://doi.org/10.1007/978-1-4615-2556-1_73
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DOI: https://doi.org/10.1007/978-1-4615-2556-1_73
Publisher Name: Springer, Boston, MA
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Online ISBN: 978-1-4615-2556-1
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