Abstract
Lyme disease is caused by the spirochete Borrelia burgdorferi (B.burgdorferi) and to date the most common vector borne infectious disease of the temperate climate 1. The multifacetal nature of the illness which affects skin, joints, nervous system and heart as well as the inability of many patients to control infection, in spite of their specific immune responses, has been a considerable challenge for clinicians and scientists1. Two aspects of B.burgdorferi infections, i.e., the pathogenesis of Lyme disease with its chronic course of tissue destruction and the role of the immune response in protection, have attracted attention in the past.
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Honarvar, N. et al. (1994). Borrelia Burgdorferi Infection in Mice: Aspects of Inflammation and Immune Responses. In: Axford, J.S., Rees, D.H.E. (eds) Lyme Borreliosis. NATO ASI Series, vol 260. Springer, Boston, MA. https://doi.org/10.1007/978-1-4615-2415-1_30
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DOI: https://doi.org/10.1007/978-1-4615-2415-1_30
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