Skip to main content

Cardiac Oedema and Physical Stress

  • Chapter
Stress and Heart Disease

Part of the book series: Developments in Cardiovascular Medicine ((DICM,volume 45))

  • 66 Accesses

Abstract

When a mechanical load is put upon one of the hearts’ ventricles, the ventricle undergoes hypertrophy. There are, however, limits to the process. In the first place there must be limits to the size to which a ventricle can grow. But, as originally pointed out by Meerson (1), there are, in addition, more subtle limits to the duration of time over which hypertrophy may be sustained. Ata certain point in the progression of the disease, the patient develops an increase in venous pressure and oedema. And then he is said to be in “cardiac failure.”

This is a preview of subscription content, log in via an institution to check access.

Access this chapter

Chapter
USD 29.95
Price excludes VAT (USA)
  • Available as PDF
  • Read on any device
  • Instant download
  • Own it forever
eBook
USD 169.00
Price excludes VAT (USA)
  • Available as EPUB and PDF
  • Read on any device
  • Instant download
  • Own it forever
Softcover Book
USD 219.99
Price excludes VAT (USA)
  • Compact, lightweight edition
  • Dispatched in 3 to 5 business days
  • Free shipping worldwide - see info
Hardcover Book
USD 219.99
Price excludes VAT (USA)
  • Durable hardcover edition
  • Dispatched in 3 to 5 business days
  • Free shipping worldwide - see info

Tax calculation will be finalised at checkout

Purchases are for personal use only

Institutional subscriptions

Preview

Unable to display preview. Download preview PDF.

Unable to display preview. Download preview PDF.

References

  1. Meerson FZ: The myocardium in hyperfunction, hypertrophy and heart failure. Circ Res (24–25) Suppl II: 1969.

    Google Scholar 

  2. Braunwald E: Heart failure: pathophysiology and treatment. Amer Heart J (102): 486–490, 1981.

    Article  PubMed  CAS  Google Scholar 

  3. Hickman JB, Cargill WH: Effect of exercise cardiac output and pulmonary arterial pressure in normal persons and inpatients with cardiovascular disease and pulmonary emphysema. J Clin Invest (27): 10–23, 1948.

    Article  Google Scholar 

  4. Merrill AJ: Edema and decreased renal blood flow in patients with congestive heart failure: evidence of ‘forward failure’ as the primary cause of edema. J Clin Invest (25): 389–400, 1946.

    Article  Google Scholar 

  5. Harris P: Evolution and the cardiac patient. Cardiovasc Res (17): 313–319, 373–378, 437–445, 1983.

    Article  PubMed  CAS  Google Scholar 

  6. Hilton SM, Spyer KM: Central nervous regulation of vascular resistance. Ann Rev Physiol (42): 399–411, 1980.

    Article  CAS  Google Scholar 

  7. Ganong WF: Neuroendocrine responses to injury and shock. Advances in Physiological Science. Volume 26. In: Bivo Zs, Kovach AGB, Spitzer JJ, Stoner HE (eds) Homeostasis in injury and shock. Pergamon, Budapest, 1981, pp. 35–44.

    Google Scholar 

Download references

Authors

Editor information

Editors and Affiliations

Rights and permissions

Reprints and permissions

Copyright information

© 1985 Martinus Nijhoff Publishing, Boston

About this chapter

Cite this chapter

Harris, P. (1985). Cardiac Oedema and Physical Stress. In: Beamish, R.E., Singal, P.K., Dhalla, N.S. (eds) Stress and Heart Disease. Developments in Cardiovascular Medicine, vol 45. Springer, Boston, MA. https://doi.org/10.1007/978-1-4613-2587-1_26

Download citation

  • DOI: https://doi.org/10.1007/978-1-4613-2587-1_26

  • Publisher Name: Springer, Boston, MA

  • Print ISBN: 978-1-4612-9622-5

  • Online ISBN: 978-1-4613-2587-1

  • eBook Packages: Springer Book Archive

Publish with us

Policies and ethics