Abstract
Brain changes occur in the newborn suffering insufficient blood oxygenation at birth. Inadequate oxygenation of the brain may cause different types of lesions, depending on the nature and duration of the stress as well as the degree of maturity of the fetus. If anoxia affects babies 37 weeks or older, lesions caused by asphyxia will be observed in the cortex or subcortical area of the brain. In premature babies, asphyxia may create lesions in the periventricular area and the matrix. Interestingly, the immature brain is more resistant to asphyxia than the adult brain, but the reason for this difference is unknown.
This is a preview of subscription content, log in via an institution.
Buying options
Tax calculation will be finalised at checkout
Purchases are for personal use only
Learn about institutional subscriptionsPreview
Unable to display preview. Download preview PDF.
Author information
Authors and Affiliations
Rights and permissions
Copyright information
© 1987 Plenum Publishing Corporation
About this chapter
Cite this chapter
Lacoius-Petruccelli, A. (1987). Physiopathology of Asphyxia and Its Staging. In: Perinatal Asphyxia. Springer, Boston, MA. https://doi.org/10.1007/978-1-4613-1807-1_3
Download citation
DOI: https://doi.org/10.1007/978-1-4613-1807-1_3
Publisher Name: Springer, Boston, MA
Print ISBN: 978-1-4612-9011-7
Online ISBN: 978-1-4613-1807-1
eBook Packages: Springer Book Archive