Abstract
The activation of the coagulation mechanisms plays an important role in the sequence of events triggered by humoral immunological factors leading to glomerular damage. In this context, wide epithelial proliferation -affecting a 50% or more of the glomeruli- and some diffuse proliferative nephritis secondary to collagen or systemic diseases are situations where the intraglomerular fibrin formation is currently agreed.
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© 1989 Kluwer Academic Publishers
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Mauri, J.M. et al. (1989). Defibrination with Ancrod in Crescentic Glomerular Lesions: Preliminary Observation. In: Andreucci, V.E., Dal Canton, A. (eds) Current Therapy in Nephrology. Developments in Nephrology, vol 24. Springer, Boston, MA. https://doi.org/10.1007/978-1-4613-0865-2_6
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DOI: https://doi.org/10.1007/978-1-4613-0865-2_6
Publisher Name: Springer, Boston, MA
Print ISBN: 978-1-4612-8209-9
Online ISBN: 978-1-4613-0865-2
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