Abstract
The relationship of selenium to the sudden infant death syndrome (SIDS) has been evaluated only summarily. Findings in the animal studies done by Money1 led him to suggest that selenium deficiency could be a causative factor in reported human cases. A study by Rhead et al.2 failed to support this contention when whole blood selenium levels were evaluated in 12 SIDS cases and compared to 4 control infants. Rhead’s report has been cited as evidence refuting selenium’s role in SIDS. We feel, nonetheless, that this small number of observations weakens the rebuttal of Money’s hypothesis.
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References
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© 1988 Plenum Press, New York
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Valentine, J., Faraji, B., Akashi, K. (1988). Selenium and Glutathione Peroxidase in Mothers Experiencing Sudden Infant Death Syndrome. In: Hurley, L.S., Keen, C.L., Lönnerdal, B., Rucker, R.B. (eds) Trace Elements in Man and Animals 6. Springer, Boston, MA. https://doi.org/10.1007/978-1-4613-0723-5_5
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DOI: https://doi.org/10.1007/978-1-4613-0723-5_5
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