Abstract
Research conducted over a period of fifty years, both in a clinical setting with human subjects, as well as in experiments with animals, has shown that plasma ceruloplasmin levels increase during periods of stress, inflammatory disease and chronic infection. However, there is little known about the factors which influence ceruloplasmin synthesis. The physiological role for the increase in circulating levels of this acute phase protein is equally obscure. The plethora of hormonal changes that precede and/or accompany these conditions suggest a variety of signals contribute to the phenomenon.
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Cousins, R.J., Barber, E.F. (1987). Regulation of Ceruloplasmin Synthesis by Retinoic Acid and Interleukin-1. In: Sorenson, J.R.J. (eds) Biology of Copper Complexes. Experimental Biology and Medicine, vol 16. Humana Press. https://doi.org/10.1007/978-1-4612-4584-1_2
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