Abstract
In his presidential address to the American Psychological Association three decades ago, Paul Meehl (1962) proposed a heuristic model for the pathogenesis of schizophrenia that anticipated the widely endorsed diathesis-stress or vulnerability models of the disorder (Hartmann et al., 1984; Zubin, Magaziner, & Steinhauer, 1983; Zubin & Spring, 1977). In typical Meehlian fashion, his provocative and now classic speech suggested that the basic pathophysiology of schizophrenia was an inherited neural integrative defect, termed schizotaxia, which interacted with virtually any known type of social learning history to produce a personality organization called, following Rado (1956), the schizotype. A small minority of schizotypes, perhaps 10% (Meehl, 1989, 1990), were theorized to decompensate into clinical schizophrenia as a result of unfavorable polygenic potentiators (e.g., high anxiety readiness, low stress resistance) and adverse life experiences (e.g., schizophrenogenic mother who is highly ambivalent and hostile). To facilitate the identification of compensated or “non-disintegrated” schizotypes, Meehl (1964) developed a richly descriptive checklist of schizotypic signs, symptoms, and traits that could be used for purposes of clinical assessment and research.
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Edell, W.S. (1995). The Psychometric Measurement of Schizotypy Using the Wisconsin Scales of Psychosis Proneness. In: Miller, G.A. (eds) The Behavioral High-Risk Paradigm in Psychopathology. Series in Psychopathology. Springer, New York, NY. https://doi.org/10.1007/978-1-4612-4234-5_1
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