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Tick Paralyses: Pathogenesis and Etiology

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Advances in Disease Vector Research

Part of the book series: Advances in Disease Vector Research ((VECTOR,volume 8))

Abstract

As has already been reviewed (33, 60), certain tick species or populations and strains of individual species are potentially capable of causing pathological and/or pathophysiological changes in humans and animals by inoculating noninfectious noxious substances during the repletion process. These substances are generally considered to be toxins. On the basis of different clinical manifestations eight forms of toxicosis may be distinguished. The most important of these toxicoses for veterinary as well as for human medicine are the tick paralyses, which basically differ both etiologically and clinically from tick-borne infections and notably from virusmediated encephalitides.

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Gothe, R., Neitz, A.W.H. (1991). Tick Paralyses: Pathogenesis and Etiology. In: Harris, K.F. (eds) Advances in Disease Vector Research. Advances in Disease Vector Research, vol 8. Springer, New York, NY. https://doi.org/10.1007/978-1-4612-3110-3_7

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