Abstract
Unlike an infectious disease associated with the presence of a particular etiologic agent, pelvic inflammatory disease (PID) is a syndrome or constellation of symptoms that can result from multiple etiologic pathways. Approximately 75% of PID is associated with progression of an initial infection from various sexually transmitted disease (STD) pathogens in the lower genital tract into the upper genital tract.1 In cases of PID not associated with an initial STD, the initial exposure may involve a mechanical insult (e.g., traumatic birth, abortion, and IUD insertion) that may result in a perturbation of the normal flora of the vagina, or a disruption such that the normal flora of the lower genital tract flora ascends into the en-dometrial cavity or fallopian tubes.2 Of course it is possible that this pathogenic mechanism may also occur from disturbances caused by STD infections.2
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Padian, N.S., Washington, A.E. (1997). Risk Factors for Pelvic Inflammatory Disease and Associated Sequelae. In: Landers, D.V., Sweet, R.L. (eds) Pelvic Inflammatory Disease. Springer, New York, NY. https://doi.org/10.1007/978-1-4612-0671-2_2
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DOI: https://doi.org/10.1007/978-1-4612-0671-2_2
Publisher Name: Springer, New York, NY
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