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Treatment by Human Fetal Neuronal Tissue Transplant at a Heterotopic Site in the Axilla in Case of Motor Neuron Disease: A Report of Two Cases

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Human Fetal Tissue Transplantation

Abstract

Motor neuron disease (MND) includes a group of diseases that affect motor neurons of the spinal cord. The spinal cord being the sole communication link between the brain and parts below the neck, any injury due to any etiology from autoimmune to excitotoxic affecting the spinal cord invariably results in dreaded sensory and motor neuronal and its loss of control. Mankind’s knowledge in the field and recognition of spinal cord injury and its consequences date back to 2500 B.C. as is evident from vivid descriptions given in Greek Papyrus archival material. Despite such early recognition, pathophysiology and prospective therapies affecting the spinal cord or its different constituents and their coordination were not identified until the early part of previous century.

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Acknowledgment

The Department of Science and Technology, Government of West Bengal, supported the investigator with a research grant during his tenure at Bijoygarh State Hospital from 1999 to 2006. The work started at Bijoygarh Government Hospital (1999–2006) and was followed up at Vidyasagore Government Hospital subsequently. The author gratefully acknowledges the support of the patients who volunteered for this research work. The guidance of Prof. K. L. Mukherjee of Biochemistry; Prof. M. K. Chhetri, former Director of Health Services; and Dr. Abhijeet Chaudhuri, Lead Neurologist, Essex Centre for Neurological Sciences, Romford, UK, are also acknowledged.

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Correspondence to Niranjan Bhattacharya D.Sc., M.D., M.S., FACS (USA) .

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Bhattacharya, N. (2013). Treatment by Human Fetal Neuronal Tissue Transplant at a Heterotopic Site in the Axilla in Case of Motor Neuron Disease: A Report of Two Cases. In: Bhattacharya, N., Stubblefield, P. (eds) Human Fetal Tissue Transplantation. Springer, London. https://doi.org/10.1007/978-1-4471-4171-6_25

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  • DOI: https://doi.org/10.1007/978-1-4471-4171-6_25

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