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Stress Urinary Incontinence: New Concept of Pathogenesis and Treatment by Pudendal Canal Decompression

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Bladder Disease, Part A

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Abstract

Genuine stress urinary incontinence (SUI), or the involuntary loss of urine through the intact urethra without a detrusor contraction during physical effort, is a common distressing condition in women. Its etiology is unknown. Many theories have been put forward, but not one is entirely satisfactory. The etiology has been related to loss of the urethrovesical angle and alteration of the urethral axis, loss of urethral length, and changes in urethra and vesical pressures.12,23 Hodgkinson13 ascribed SUI to the internal urinary meatus, forced by straining into a dependent position, so that the urethra becomes unable to resist the hydraulic effects of the intravesical fluid column in this position. Hutch15 suggested that the lowered position of the base plate resulted in a partially open internal meatus, thus lowering the urethral resistance to stress.

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Authors and Affiliations

  1. Department of Surgery and Experimental Research, Cairo University Faculty of Medicine, Cairo, Egypt

    Ahmed Shafik

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  1. Ahmed Shafik
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Editors and Affiliations

  1. Children’s Hospital and Harvard Medical School, Boston, Massachusetts, USA

    Anthony Atala

  2. Ridgefield, Connecticut, USA

    Debra Slade

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Shafik, A. (2003). Stress Urinary Incontinence: New Concept of Pathogenesis and Treatment by Pudendal Canal Decompression. In: Atala, A., Slade, D. (eds) Bladder Disease, Part A. Springer, Boston, MA. https://doi.org/10.1007/978-1-4419-8889-8_29

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  • DOI: https://doi.org/10.1007/978-1-4419-8889-8_29

  • Publisher Name: Springer, Boston, MA

  • Print ISBN: 978-1-4613-4707-1

  • Online ISBN: 978-1-4419-8889-8

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