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The Canonical and Unconventional Ligands of the Herpesvirus Entry Mediator

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Advances in TNF Family Research

Part of the book series: Advances in Experimental Medicine and Biology ((AEMB,volume 691))

Abstract

The maintenance of immune homeostasis requires a balance between stimulatory and inhibitory pathways. The herpesvirus entry mediator (HVEM; TNFRSF14) [1] serves as one of two key entry routes used by herpes simplex virus-1 and herpes simplex virus-2 to infect cells [2].

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Acknowledgments

Support for this work was provided by grants from the National Institutes of Health (R37AI33068, AI067890, AI048073, and CA069381).

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Correspondence to Timothy C. Cheung or Carl F. Ware .

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Cheung, T.C., Ware, C.F. (2011). The Canonical and Unconventional Ligands of the Herpesvirus Entry Mediator. In: Wallach, D., Kovalenko, A., Feldmann, M. (eds) Advances in TNF Family Research. Advances in Experimental Medicine and Biology, vol 691. Springer, New York, NY. https://doi.org/10.1007/978-1-4419-6612-4_36

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