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Distribution of Pelvic and Abdominal Adenomyomas

  • Ronald E. Batt
Chapter

Abstract

By 1920, Cullen accepted three models for the pathogenesis of extrauterine adenomyomas: (1) from embryonic müllerian rests, (2) “springing from” the posterior wall of the cervix or body of the uterus and invading the rectum, and (3) from overflow of uterine mucosa – shed from an adenomyoma of the rectovaginal septum onto tube and ovary – the same flowing metaphor that he used to explain the pathogenesis of diffuse uterine adenomyomas. Cullen came tantalizingly close to – and yet so far from – Sampson’s later observations when he wrote: “One gathers the impression that the uterine mucosa from the diffuse adenomyoma on the posterior surface of the cervix and uterus has overflowed upon the adherent tube [and ovary].”1 Cullen had the implantation pathogenesis in reverse.

Keywords

Sigmoid Colon Uterine Horn Uterosacral Ligament Johns Hopkins Hospital Broad Ligament 
These keywords were added by machine and not by the authors. This process is experimental and the keywords may be updated as the learning algorithm improves.

Copyright information

© Springer-Verlag LondonLimited 2011

Authors and Affiliations

  • Ronald E. Batt
    • 1
  1. 1.State University of New York at BuffaloBuffaloUSA

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