Advertisement

Ischemic and Visceral Pain

  • Robby Romero
  • Dmitri Souzdalnitski
  • Trevor Banack
Chapter

Abstract

Ischemic pain is a distinct type of pain associated with decreased or complete cessation of blood flow rather than direct damage to tissues or neuropathic processes. The variety of pathological conditions which produce arterial or venous blood flow obstruction will be discussed. Ischemic pain is often intractable and requires acute intervention aimed at reperfusion of the ischemic tissues. This chapter describes various pain management tools which may be utilized for treatment of patients with chronic ischemic pain as adjuncts to reperfusion techniques for the most common condition associated with ischemic pain, peripheral arterial occlusive disease.

Keywords

Spinal Cord Stimulation Critical Limb Ischemia Visceral Pain Peripheral Arterial Occlusive Disease Peripheral Nerve Block 
These keywords were added by machine and not by the authors. This process is experimental and the keywords may be updated as the learning algorithm improves.

References

  1. Bonica JJ. General consideration of acute Pain. In: Bonica JJ, editor. The management of Pain. Philadelphia, PA: Lea & Febiger; 1990a. pp. 159–79.Google Scholar
  2. Bonica JJ. Applied anatomy relevant to Pain. In: Bonica JJ, editor. The management of pain. Philadelphia, PA: Lea & Febiger; 1990b. pp. 133–58.Google Scholar
  3. Bonica JJ. General considerations of abdominal pain. In: Bonica JJ, editor. The management of pain. Philadelphia, PA: Lea & Febiger; 1990c. pp. 1146–85.Google Scholar
  4. Cervero F. Sensory innervation of the viscera: peripheral basis of visceral pain. Physiol Rev. 1994;74:95–138.PubMedGoogle Scholar
  5. Dubé GR, Elagoz A, Mangat H. Acid sensing ion channels and acid nociception. Curr Pharm Des. 2009;15(15):1750–66.PubMedCrossRefGoogle Scholar
  6. Foletti A, Durrer A, Buchser E. Neurostimulation technology for the treatment of chronic pain: a focus on spinal cord stimulation. Expert Rev Med Devices. 2007;4(2):201–14.PubMedCrossRefGoogle Scholar
  7. Giamberberardino MS, Vecchiet L. Pathophysiology of visceral pain. Curr Pain Headache Rep. 1997;23–33.Google Scholar
  8. Gofeld M, Faclier G. Bilateral pain relief after unilateral thoracic percutaneous sympathectomy. Can J Anaesth. 2006;53(3):258–62.PubMedCrossRefGoogle Scholar
  9. Janig W, Morrison JFB. Functional properties of spinal visceral afferent supplying abdominal and pelvic organs with special emphasis on visceral nociception. In: Cervero F, Morrison JFB, editors. Visceral sensation progress in brain research. Amsterdam: Elsevier; 1986. pp. 87–114.CrossRefGoogle Scholar
  10. Willert RP, et al. The development and maintenance of human visceral pain hypersensitivity is dependent on the n-methyl-d-aspartate receptor. Gastroenterology 2004;126;683–92.PubMedCrossRefGoogle Scholar

Copyright information

© Springer Science+Business Media, LLC 2011

Authors and Affiliations

  • Robby Romero
    • 1
  • Dmitri Souzdalnitski
    • 2
  • Trevor Banack
    • 2
  1. 1.Yale University School of MedicineNew HavenUSA
  2. 2.Department of AnesthesiologyYale University School of MedicineNew HavenUSA

Personalised recommendations