Abstract
Noxious thermal, mechanical, or chemical stimuli evoke pain through excitation of the peripheral terminals called nociceptors.1,2 Many kinds of ionotropic and metabotropic receptors are involved in this process.3ȁ TRPV1, a capsaicin receptor, is a nociceptor-specific ion channel that serves as the molecular target of capsaicin, having six transmembrane domains with a short hydrophobic stretch between the fifth and sixth transmembrane domains.7
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Tominaga, M., Moriyama, T. (2007). Functional Interaction Between ATP and TRPV1 Receptors. In: Wang, D.H. (eds) Molecular Sensors for Cardiovascular Homeostasis. Springer, Boston, MA. https://doi.org/10.1007/978-0-387-47530-1_7
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DOI: https://doi.org/10.1007/978-0-387-47530-1_7
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