Abstract
The view that genetic mechanisms underlie many forms of epilepsy is widely accepted.1 Thus, genetically-derived models of epilepsy can serve as important vehicles to study the mechanisms of epileptogenesis,2 particularly if they can isolate underlying factors that are uncontaminated by a recent history of seizure activity.
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McIntyre, D.C., Gilby, K.L. (2005). Electrophysiological and Anatomical Differences, Behavioral Comorbidities and Gene Expression in Fast and Slow Kindling Rat Strains. In: Corcoran, M.E., Moshé, S.L. (eds) Kindling 6. Advances in Behavioral Biology, vol 55. Springer, Boston, MA. https://doi.org/10.1007/0-387-26144-3_17
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