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A Case of False Foster-Kennedy Syndrome

  • Xiaobin Xie
  • Ning Fan
  • Ningli Wang
Chapter
Part of the Advances in Visual Science and Eye Diseases book series (AVSED, volume 2)

Abstract

The etiology of Foster-Kennedy syndrome, also known as Kennedy’s syndrome or basilar frontal lobe syndrome, involves space-occupying lesions at the basilar part of the frontal lobe, such as abscess, hemangioma, meningioma of the sphenoid ridge, carotid atherosclerosis, arachnoiditis, and brain trauma. In typical Foster-Kennedy syndrome cases, unilateral tumor in the anterior cranial fossa compresses the ipsilateral optic nerve, resulting in ipsilateral optic atrophy, and meanwhile space-occupying lesions induce intracranial hypertension, leading to contralateral optic disc edema. The patient in the case below suffered from Foster-Kennedy syndrome with optic disc edema in one eye and a pale optic disc in the other. What is the mechanism behind this condition?

References

  1. 1.
    Acebes X, Arruga J, Acebes JJ, et al. Intracranial meningiomatosis causing Foster Kennedy syndrome by unilateral optic nerve compression and blockage of the superior sagittal sinus. J Neuroophthalmol. 2009;29(2):140–2.CrossRefGoogle Scholar
  2. 2.
    Bansal S, Dabbs T, Long V. Pseudo-Foster Kennedy syndrome due to unilateral optic nerve hypoplasia: a case report. J Med Case Rep. 2008;2(1):1–2.CrossRefGoogle Scholar

Copyright information

© Springer Nature Singapore Pte Ltd. & People's Medical Publishing House, PR of China 2019

Authors and Affiliations

  • Xiaobin Xie
    • 1
  • Ning Fan
    • 2
  • Ningli Wang
    • 3
  1. 1.Eye Hospital of China Academy of Chinese Medical SciencesBeijingChina
  2. 2.Shenzhen Eye Hospital, Shenzhen UniversityShenzhenChina
  3. 3.Department of OphthalmologyBeijing Tongren Hospital, Capital Medical UniversityBeijingChina

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