Abstract
Legg-Calvé-Perthes disease (LCPD) represents a type of idiopathic avascular necrosis of the femoral head which typically develops during childhood [1, 2]. Its etiology is considered to be multifactorial but is generally unknown; genetic, constitutional, and environmental factors have been implicated [3]. Despite receiving treatment, some patients initially develop morphological changes in the femoral head and secondarily acetabular deformity, femoroacetabular in congruency, and the abnormal hip movement that could lead to early-onset secondary osteoarthritis [1, 4]. In a current prospective cohort, 5% of LCPD patients underwent THA at 20 years after nonoperative treatment [5]. Stulberg classification is used as prognostic tool of hip osteoarthritis at skeletal maturity [2]; Stulberg types III–V are more susceptible to the development of early secondary hip osteoarthritis [1].
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Kenanidis, E. et al. (2018). THA Following Pediatric Hip Diseases (Legg-Calvé-Perthes/Slipped Capital Femoral Epiphysis). In: Tsiridis, E. (eds) The Adult Hip - Master Case Series and Techniques. Springer, Cham. https://doi.org/10.1007/978-3-319-64177-5_9
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