Abstract
Ventricular septal defects (VSDs) are the most common congenital heart disease, accounting for 25 % of all congenital heart defects [1]. Alternatively, a VSD can be acquired during adulthood either after a myocardial infarction (MI), as a complication of cardiac surgery, or rarely after trauma to the chest. VSDs secondary to MI are much less common in the post-reperfusion therapy era, occurring in only 0.2–0.34 % of patients receiving thrombolysis for acute MI in the Global Utilization of Streptokinase and Tissue Plasminogen Activator for Occluded Coronary Arteries (GUSTO-I) trial [2].
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Pighi, M., Asgar, A.W. (2017). Difficult Cases and Complications from the Catheterization Laboratory: Postinfarction Ventricular Septal Defect Closure. In: Reimers, B., Moussa, I., Pacchioni, A. (eds) Percutaneous Interventions for Structural Heart Disease. Springer, Cham. https://doi.org/10.1007/978-3-319-43757-6_27
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DOI: https://doi.org/10.1007/978-3-319-43757-6_27
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