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Abstract

Metabolic and tubular defects occurring after kidney transplantation are multifactorial and depend on 1) the degree of efficiency of the transplanted organ, 2) side effects of immunosuppressive drugs, 3) late effects of dialysis treatment (e.g., amyloidosis, bone changes), 4) coexistent diseases, often accompanying the kidney failure (e.g., HBV or HCV infection), and 5) consequences of acute and chronic rejections, which by themselves are inflammatory processes.

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Wadi N. Suki M.D. Shaul G. Massry M.D.

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Gradowska, L., Paczek, L. (1998). Tubular and Metabolic Dysfunction Following Transplantation. In: Suki, W.N., Massry, S.G. (eds) Suki and Massry’s THERAPY OF RENAL DISEASES AND RELATED DISORDERS. Springer, Boston, MA. https://doi.org/10.1007/978-1-4757-6632-5_66

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