Abstract
Seropositive or antibody-transferred kittens have been reported to have an overt disease after parenteral challenge with virulent feline infectious peritonitis (FIP) virus, suggesting that the effusive type FIP2,3 might result from an interaction between the virus and host response. On the other hand, feline leukemia virus infection was suggested to enhance the infection of FIP virus1. The present study deals with roles of circulating antibodies as well as thymus-dependent lymphocytes in pathogenesis after oral infection of FIP virus.
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References
S. M. Cotter, C. E. Gilmore, and C. Rollins. Multiple cases of feline leukemia and feline infectious peritonitis in a household, J.Am.Vet.Med.Assoc. 162, 1054–1058, (1973).
N. C. Pedersen, and J. F. Boyle. Immunologic phenomena in the effusive form of feline infectious peritonitis, Am.J.Vet.Res. 41, 868–876, (1980).
R. C. Weiss, and F. W. Scott. Pathogenesis of feline infectious peritonitis: Pathologic changes and immunofluorescence. Am.J.Vet.Res. 42, 2036–2048, (1980).
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© 1984 Plenum Press, New York
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Hayashi, T., Doi, K., Fujiwara, K. (1984). Role of Circulating Antibodies and Thymus-Dependent Lymphocytes in Production of Effusive Type Feline Infectious Peritonitis After Oral Infection. In: Rottier, P.J.M., van der Zeijst, B.A.M., Spaan, W.J.M., Horzinek, M.C. (eds) Molecular Biology and Pathogenesis of Coronaviruses. Advances in Experimental Medicine and Biology, vol 173. Springer, Boston, MA. https://doi.org/10.1007/978-1-4615-9373-7_38
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DOI: https://doi.org/10.1007/978-1-4615-9373-7_38
Publisher Name: Springer, Boston, MA
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