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Diastolic Relaxation of the Heart pp 217–229Cite as

Diastolic Function during Exercise-Induced Ischemia in Man

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Abstract

Diastolic pressures frequently increase dramatically during ischemia. The mechanism for the pressure increase has been the subject of many clinical and experimental studies [1–5]. Impaired relaxation has been shown in humans during pacing-induced ischemia [2]. The alterations in left ventricular pressure decay may be important in elevating diastolic pressures, producing the upward shift in the diastolic pressure-volume relation, and impairing the time course and extent of left ventricular filling.

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Authors
  1. John D. Carroll
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  2. Otto M. Hess
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  3. Hans Peter Krayenbuehl
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Editor information

Editors and Affiliations

  1. Harvard Medical School, Cardiovascular Division, Beth Israel Hospital, Boston, Massachusetts, USA

    William Grossman M.D. (Herman Dana Professor of Medicine, Chief) (Herman Dana Professor of Medicine, Chief)

  2. Harvard Medical School, Hemodynamic Research Laboratory, Beth Israel Hospital, Boston, Massachusetts, USA

    Beverly H. Lorell M.D. (Assistant Professor of Medicine, Co-Director) (Assistant Professor of Medicine, Co-Director)

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© 1987 Martinus Nijhoff Publishing

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Carroll, J.D., Hess, O.M., Krayenbuehl, H.P. (1987). Diastolic Function during Exercise-Induced Ischemia in Man. In: Grossman, W., Lorell, B.H. (eds) Diastolic Relaxation of the Heart. Springer, Boston, MA. https://doi.org/10.1007/978-1-4615-6832-2_22

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  • DOI: https://doi.org/10.1007/978-1-4615-6832-2_22

  • Publisher Name: Springer, Boston, MA

  • Print ISBN: 978-1-4615-6834-6

  • Online ISBN: 978-1-4615-6832-2

  • eBook Packages: Springer Book Archive

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