Molecular Brain

, 9:21 | Cite as

Erratum to: Novel function of PIWIL1 in neuronal polarization and migration via regulation of microtubule-associated proteins

  • Ping-ping Zhao
  • Mao-jin Yao
  • Si-yuan Chang
  • Lan-tao Gou
  • Mo-fang Liu
  • Zi-long Qiu
  • Xiao-bing Yuan
Open Access
Erratum
  • 585 Downloads
After publication of this article [1], the authors noticed two errors in Figs. 2e and 3e.
Fig. 2

PIWIL1 is required for the multipolar–bipolar transition of postmitotic neurons. a, d Morphology of labeled neurons in different cortical regions 3 or 5 days post-IUE with siRNA 1. b, e Traces of labeled neurons 3 or 5 days after IUE respectively. c, f Percentage of bipolar cells (white arrows) in different cortical regions. Data are from at least 3 independent IUE experiments. g Typical morphology of labeled mouse neurons in the IZ 3 days after IUE with RNAi 2 or RNAi 2 plus HIWI compared with individual control plasmid. h Percentage of bipolar cells at the IZ of electroporated mouse cortex. Scale bar, 30 μm. Error bar, SEM, **P < 0.01, ***P < 0.001 (Student’s t-test)

Fig. 3

PIWIL1 knockdown impairs polarization of cortical neurons ex vivo. a Diagram of the ex vivo assay. b, c Average numbers of primary neurites of electroporated cells. d Immunostaining: cultured neurons with PIWIL1 knockdown exhibited multipolar morphology and lower levels of Tau but not Tuj1. e Average neurites’ fluorescence intensity of Tau in GFP+ neurons. Scale bar, 20 μm. Error bar, SEM, *P < 0.05, ***P < 0.001 (Student’s t-test)

The images provided for Fig. 2e in squares ‘Scramble upCP’ and ‘Scramble IoCP’ were incorrect. The correct version of Fig. 2 is included in this erratum.

In Fig. 3e, the label ‘RNAi 4’ was missing from the x axis. The correct version of Fig. 3 is also included in this erratum.

Reference

  1. 1.
    Zhao P et al. Novel function of PIWIL1 in neuronal polarization and migration via regulation of microtubule-associated proteins. Mol Brain. 2015;8:39. doi: 10.1186/s13041-015-0131-0.PubMedCentralCrossRefPubMedGoogle Scholar

Copyright information

© Zhao et al. 2016

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

Authors and Affiliations

  • Ping-ping Zhao
    • 1
    • 2
  • Mao-jin Yao
    • 1
    • 2
  • Si-yuan Chang
    • 1
    • 2
  • Lan-tao Gou
    • 2
    • 3
  • Mo-fang Liu
    • 3
  • Zi-long Qiu
    • 1
  • Xiao-bing Yuan
    • 1
    • 4
  1. 1.Institute of Neuroscience and State Key Laboratory of Neuroscience, Shanghai Institutes for Biological SciencesChinese Academy of SciencesShanghaiChina
  2. 2.Graduate School of Chinese Academy of SciencesShanghaiChina
  3. 3.State Key Laboratory of Molecular Biology, Institute of Biochemistry and Cell Biology, Shanghai Institutes for Biological SciencesChinese Academy of SciencesShanghaiChina
  4. 4.Current Affiliation: Hussman Institute for AutismBaltimoreUSA

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