Table 3 Comparison of the present study with previous studies, showing the results at the end of the experiment

From: Long-term exposure to high-concentration silver nanoparticles induced toxicity, fatality, bioaccumulation, and histological alteration in fish (Cyprinus carpio)

Fish NPs type Concentration selection method Duration Organs analyzed Histological study Bioaccumulation Reference
Common carp fish AgNPs 0.04, 0.08 mg/L 96 h Gills, liver, and intestine Epithelial lifting, telangiectasia in secondary lamellae, and epithelial necrosis Mostly in the liver and intestine [27]
Zebrafish, rainbow trout fish CuNPs 1 mg/L 96 h Gills Damage of gill filaments N/A [28]
Epinephelus coioides CuNPs 100 μg/L 25 days Gills, intestine, liver, and muscles Cell apoptosis in gills, liver Liver, gills, intestine, and muscles [29]
Common carp fish AgNPs 0.75 mg/L 14 days Gills Telangiectasia and epithelial cell hyperplasia in fish gills N/A [5]
Common carp fish ZnONPs 50 mg/L 30 days Gills, liver intestine, and brain Irregular cell outline, abnormal pyknotic nuclei, shrinkage of cell Mostly accumulated in the liver and gills [30]
Common carp fish CuNPs N/A 21 days Gills, intestine, and liver N/A Mostly accumulated in the liver and intestine [31]
Common carp fish B-AgNPs 0.03, 0.06, 0.09 mg/L 20 days Gills, intestine, muscles, liver Histological alteration mostly occurred at the highest concentration of B-AgNPs in gills and intestine Widely accumulated in the liver, followed by the intestine, gills, and muscle Our study