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Correction to: Inhaled gold nanoparticles cause cerebral edema and upregulate endothelial aquaporin 1 expression, involving caveolin 1 dependent repression of extracellular regulated protein kinase activity

  • Ching-Yi Chen
  • Po-Lin Liao
  • Chi-Hao Tsai
  • Yen-Ju Chan
  • Yu-Wen Cheng
  • Ling-Ling Hwang
  • Kuan-Hung Lin
  • Ting-Ling Yen
  • Ching-Hao LiEmail author
Open Access
Correction

Correction to: Part Fibre Toxicol

https://doi.org/10.1186/s12989-019-0324-2

It was highlighted that the original article [1] contained the wrong Fig. 1. This Correction article shows the correct Fig. 1. The original article has been updated.
Fig. 1

Au-NPs induced aquaporin-1 (AQP1) protein expression in bEnd.3 cells. a/b The bEnd.3 cells (an immortalized mouse cerebral endothelial cell line) were exposed to Au-NPs (or Au-MPs) and the expression level of AQP1 was detected by western blots. Representative images showed an increase of AQP1 protein level in Au-NP-treated groups, whereas AQP1 protein level remained unaffected in Au-MP-treated groups. a concentration-dependent treatment; cells were incubated with 10, 50, 100 and 500 ng/mL Au-NPs for 24 h. b time-dependent treatment; cells were incubated with 500 ng/mL Au-NPs for 3, 6, 12, and 24 h. (* p < 0.05, ** p < 0.01, and *** p < 0.001 indicates statistically significant difference from the control group; N = 11). c Representative images of immunofluorescent staining, the Au-NP-induced AQP1 and the nucleus was manifested by red and blue fluorescence, respectively. A gain of red fluorescence in cell membrane and cytosol was observed in Au-NP-treated bEnd.3 cells (500 ng/mL; 24 h), as compared to control. d Transendothelial permeability assay was performed as described in Materials and Methods. Au-NP treatment (500 ng/mL; 24 h) made bEnd.3 cell more permeable to water. (* p < 0.05, indicates statistically significant difference from the control group; N = 12)

Reference

Copyright information

© The Author(s). 2019

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

Authors and Affiliations

  • Ching-Yi Chen
    • 1
    • 2
    • 3
  • Po-Lin Liao
    • 3
    • 4
  • Chi-Hao Tsai
    • 4
  • Yen-Ju Chan
    • 1
    • 2
  • Yu-Wen Cheng
    • 3
  • Ling-Ling Hwang
    • 1
    • 2
  • Kuan-Hung Lin
    • 5
  • Ting-Ling Yen
    • 6
  • Ching-Hao Li
    • 1
    • 2
    Email author
  1. 1.Department of Physiology, School of Medicine, College of MedicineTaipei Medical UniversityTaipeiTaiwan
  2. 2.Graduate Institute of Medical Sciences, College of MedicineTaipei Medical UniversityTaipeiTaiwan
  3. 3.School of PharmacyTaipei Medical UniversityTaipeiTaiwan
  4. 4.Institute of Food Safety and Health Risk Assessment, School of Pharmaceutical SciencesNational Yang-Ming UniversityTaipeiTaiwan
  5. 5.Institute of Biomedical Sciences, Mackay Medical CollegeNew Taipei CityTaiwan
  6. 6.Department of Medical Research, Cathay General HospitalTaipeiTaiwan

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