Advertisement

Neurocritical Care

, Volume 28, Issue 3, pp 402–403 | Cite as

Response to Letter: Mechanisms Leading to Stress Cardiomyopathy Following Neurologic Injury Remain Elusive

  • Kevin T. Gobeske
  • Eelco F. Wijdicks
Letter to the Editor
  • 76 Downloads

Notes

Compliance with Ethical Standards

Conflicts of interest

The authors declare that they have no conflict of interest.

References

  1. 1.
    Wittstein IS, Thiemann DR, Lima JA, Baughman KL, Schulman SP, Gerstenblith G, Wu KC, Rade JJ, Bivalacqua TJ, Champion HC. Neurohumoral features of myocardial stunning due to sudden emotional stress. N Engl J Med. 2005;352(6):539–48.CrossRefPubMedGoogle Scholar
  2. 2.
    Akashi YJ, Goldstein DS, Barbaro G, Ueyama T. Takotsubo cardiomyopathy: a new form of acute, reversible heart failure. Circulation. 2008;118(25):2754–62.CrossRefPubMedPubMedCentralGoogle Scholar
  3. 3.
    Lyon AR, Rees PS, Prasad S, Poole-Wilson PA, Harding SE. Stress (Takotsubo) cardiomyopathy—a novel pathophysiological hypothesis to explain catecholamine-induced acute myocardial stunning. Nat Clin Pract Cardiovasc Med. 2008;5(1):22–9.CrossRefPubMedGoogle Scholar
  4. 4.
    Paur H, Wright PT, Sikkel MB, Tranter MH, Mansfield C, O’Gara P, Stuckey DJ, Nikolaev VO, Diakonov I, Pannell L, Gong H, Sun H, Peters NS, Petrou M, Zheng Z, Gorelik J, Lyon AR, Harding SE. High levels of circulating epinephrine trigger apical cardiodepression in a β2-adrenergic receptor/Gi-dependent manner: a new model of Takotsubo cardiomyopathy. Circulation. 2012;126(6):697–706.CrossRefPubMedPubMedCentralGoogle Scholar

Copyright information

© Springer Science+Business Media, LLC, part of Springer Nature and Neurocritical Care Society 2018

Authors and Affiliations

  1. 1.Division of Neurocritical Care, Department of NeurologyMayo ClinicRochesterUSA

Personalised recommendations