Journal of Gastroenterology

, Volume 53, Issue 4, pp 576–577 | Cite as

Erratum to: Action and function of Wnt/β-catenin signaling in the progression from chronic hepatitis C to hepatocellular carcinoma

  • Wenhui Wang
  • Qiuwei Pan
  • Gwenny M. Fuhler
  • Ron Smits
  • Maikel P. Peppelenbosch
Erratum
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Erratum to: J Gastroenterol (2017) 52:419–431 DOI 10.1007/s00535-016-1299-5

In the original publication of the article, the frequency of loss-of-function mutations in AXIN1, AXIN2 and APC was underestimated in Table 1. The correct frequencies of these mutations in HCC are 10.4, 3.3, and 1.4%, respectively (in the following page).
Table 1

Genetic mutation in components of Wnt/β-catenin pathway in HCC

References

Patients

Mutant samples

Region

CTNNB1

AXIN1

AXIN2

APC

Rebouissou et al. [39]

373

146 (39%)

NA

NA

NA

France, Spain, Italy

Hirotsu et al. [40]

9

2 (22.2%)

NA

NA

NA

Japan

Schulze et al. [41]

243

95 (37.4%)

27 (11.1%)

3 (1.2%)

4 (1.6%)

France, Italy, Spain

Kan et al. [42]

88

14 (15.9%)

4 (4.5%)

2 (2.3%)

2 (2.3%)

China

Kitao et al. [43]

134

27 (20.1%)

NA

NA

NA

Japan

Ding et al. [44]

156

15 (9.6%)

NA

NA

NA

China

Tornesello et al. [45]

67

10 (14.9%)

NA

NA

NA

Southern Italy

Cleary et al. [46]

87

20 (22.9%)

NA

NA

NA

Canada, NC

Guichard et al. [47]

125

41 (32.8%)

19 (15.2%)

NA

2 (1.6%)

France

Lachenmayer et al. [48]

90

29 (32.2%)

NA

NA

NA

USA, Netherlands, Italy, Spain, Germany

Li et al. [49]

139

28 (20.1%)

NA

NA

NA

USA, Netherlands, China

Cieply et al. [50]

32

9 (28.1%)

NA

NA

NA

USA

Bengochea et al. [38]

62

16 (25.8%)

NA

NA

NA

Thailand, France

Austinat et al. [51]

40

10 (25%)

2 (5%)

NA

NA

Germany

Kim et al. [52]

36

1 (2.8%)

9 (25%)

NA

NA

Korea

Zucman-Rossi et al. [53]

45

18 (40%)

5 (11.1%)

NA

NA

France

Boyault et al. [54]

120

34 (28.3%)

13 (10.8%)

NA

NA

France

Zucman-Rossi et al. [55]

96

12 (12.5%)

NA

NA

NA

France

Park et al. [56]

81

13 (16%)

5 (6.2%)

NA

NA

Korea

Ishizaki et al. [57]

89

10 (11.2%)

13 (14.6%)

9 (10.1%)

NA

Japan

Cui et al. [58]

34

15 (44.1%)

NA

NA

NA

China

Edamoto et al. [59]

100

24 (24%)

NA

NA

0

Japan, Switzerland

Taniguchi et al. [60]

73

14 (19.2%)

7 (9.6%)

2 (2.7%)

NA

UK

Wong et al. [61]

60

7 (11.7%)

NA

NA

NA

China

Mao et al. [62]

262

37 (14.1%)

NA

NA

NA

Taiwan

Cui et al. [63]

34

15 (44.1%)

NA

NA

NA

China

Laurent-Puig et al. [64]

137

26 (19%)

12 (8.8%)

NA

NA

France

Devereux et al. [65]

62

5 (8.1%)

NA

NA

NA

China

Hsu et al. [66]

434

57 (13.1%)

NA

NA

NA

Taiwan

Satoh et al. [67]

87

0 (0%)

5 (5.7%)

NA

NA

Japan

Huang et al. [68]

22

9 (41%)

NA

NA

NA

Japan, Switzerland

Legoix et al. [69]

119

21 (17.6%)

NA

NA

NA

France

Terris et al. [70]

73

14 (19.2%)

NA

NA

NA

France

Kondo et al. [71]

38

9 (24%)

NA

NA

NA

Japan

Nhieu et al. [72]

35

12 (34.3%)

NA

NA

NA

France

Miyoshi et al. [73]

75

14 (18.7%)

NA

NA

NA

Japan

de La Coste et al. [74]

31

8 (25.8%)

NA

NA

NA

France

Total

3788

837 (22.1%)

121 (10.4%)

16 (3.3%)

8 (1.4%)

 

NA not analyzed

On page 421, in the section of “Aberrant activation of Wnt/β-catenin signaling during HCC”, the sentence following “Missense, insertion, or partial deletions within CTNNB1 exon 3 lead to … stabilization of β-catenin in the cytoplasm” should be read as “Less frequently, loss-of-function mutation of AXIN1, AXIN2, or APC is found in 10.4, 3.3, and 1.4% of HCCs respectively…”.

Copyright information

© Japanese Society of Gastroenterology 2017

Authors and Affiliations

  • Wenhui Wang
    • 1
  • Qiuwei Pan
    • 1
  • Gwenny M. Fuhler
    • 1
  • Ron Smits
    • 1
  • Maikel P. Peppelenbosch
    • 1
  1. 1.Department of Gastroenterology and HepatologyErasmus University Medical CenterRotterdamThe Netherlands

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