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The health-schooling relationship: evidence from Swedish twins

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Abstract

Health and education are known to be highly correlated, but the mechanisms behind the relationship are not well understood. In particular, there is sparse evidence on whether adolescent health may influence educational attainment. Using a large registry dataset of twins, including comprehensive information on health status at the age of 18 and later educational attainment, we investigate whether health predicts final education within monozygotic (identical) twin pairs. We find no evidence of this and conclude that health in adolescence may not have an influence on the level of schooling. Instead, raw correlations between adolescent health and schooling appear to be driven by genes and twin-pair-specific environmental factors.

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Notes

  1. Birth weight divided by gestation.

  2. A few studies, including Smith (2009) and Currie et al. (2010), have examined the role of childhood health using sibling comparisons, thus not fully accounting for potential genetic confounders. As will be shown, accounting for genetic confounders is important for the interpretation of the relationship.

  3. This has previously been pointed out by Miller et al. (2005).

  4. We have used these military enlistment records in several other studies. For example, Lundborg et al. (2014b) showed the existence of a relationship between health and earnings both within families and twin pairs. Our data source is the same as the one used by Sandewall et al. (2014).

  5. Although we use the term “adolescent” throughout, it should be noted that health is a process from birth up to the current age and that our measure will reflect a sum of health shocks up to the age of 18.

  6. Up until 1972, individuals were usually called to undergo the military enlistment test at age 19, whereas in later years most individuals underwent the enlistment test at age 18.

  7. A number of studies suggest that childhood disease burden has strong effects on the growth of a person (e.g., Bozzoli et al. 2009), and a number of recent studies have used indicators of height or stunted growth to proxy for health early in life (e.g., Case and Paxson 2008; Bhalotra and Rawlings 2011). Bozzoli et al. (2009) suggest that height is related to rates of postneonatal mortality due to pneumonia and possibly congenital anomalies and intestinal disease. They find little evidence that height would be related to mortality rates for other conditions. In poorer countries, stunting has also been related to diarrhea (Martorell et al. 1975; Lutter et al. 1989).

  8. With the exception of conditions of the sensory organs; low hearing acuity and low visual acuity are instead studied separately.

  9. BMI, body mass index, is calculated as (weight in kilograms)/(height in meters)2.

  10. See Carlsson et al. (2015) for a further explanation of the subtests.

  11. To be specific, we use information on grades and results from a math test described by Pettersson (1993) and restrict attention to children with Swedish-born parents, yielding 7399 individuals. We standardize test scores based on subtest 1a and 2 (as these were taken by all children), sum these, and standardize again. Finally, the average test score is then calculated for each grade in general and special math. Each individual in our data is then assigned this average standardized test score corresponding to their grade, whether in general math or in special math.

  12. Our approach has the advantage that twin pairs with missing data will not contribute to identification of health’s effect on schooling. However, in order to take into account the interrelations between regression variables and the occurances of missings, one can also use multiple imputation (Rubin 1986). Implementing this analysis, using the STATA command mi input chained makes little difference to our results. Results are also similar if dropping individuals with missing information on any variable.

  13. More specifically, individuals are excluded if global health is missing. It is not possible to determine whether there is missing information on specific health conditions. However, for individuals reported to suffer from specific health conditions, data on global health is always available. Missing data on global health usually reflects that the data from the enlistment process has not been electronically recorded, which is the case for all conscriptions that took place in 1985 and for parts of several other years.

  14. Descriptive statistics for DZ twins are reported in Appendix Table 7. There are many significant, albeit small, differences in average characteristics when comparing the full male population sample, the MZ twin sample, and the DZ twin sample. For example, both MZ and DZ twins tend to have slightly better global health than the overall male population at age 18. We have run regressions where we re-weight the MZ and DZ twin samples to match the frequencies of global health observed in our full sample; however, this makes little difference to the results.

  15. An effect of parental education on child’s health and abilities (as measured at military enlistment in Sweden) was shown in Lundborg et al. (2014a).

  16. This is most likely because the NSA de facto raised their thresholds and evaluated individuals’ health less favorably in more recent years. Accounting for this by the use of year of birth dummies increases the effect of global health in absolute value from −0.46 to −0.67 (an increase by 46 %). Adding controls for parental education then reduces this effect by 24 %, which reduces by another 6 % when also controlling for family income.

  17. It is possible to run separate OLS regressions on twin pairs differing in global health (thus contributing to the FE estimate) and on those not differing (thus not contributing to the FE estimate). Doing this, with controls, we find an effect of −0.34 in the former sample and −0.58 in the latter. It is not surprising that the latter estimate is larger, as it relies entirely on between-pair comparisons, where healthier twin pairs are also likely to have other advantageous characteristics compared to less healthy ones. Still, it is noteworthy that the former estimate is substantial and strongly significant, as opposed to the result obtained using fixed effects.

  18. We can also control for different subscores of cognitive ability. The significant effect turns out to be driven by logical skills and synonym understanding, but whether we use the overall score or the subscores makes virtually no difference for the coefficient on health.

  19. The strong positive relationships between respiratory conditions and education in OLS models without controls may be explained by the fact that smoking used to be more common among individuals with higher levels of educational attainment, a fact that has previously been shown for the USA (Sander 1995). Using the Swedish Level of Living Survey from 1968, we regressed a smoking dummy on a dummy indicating “more than basic education” (no controls) and obtained a coefficient of 0.06; t = 4.88. With this positive relationship, in combination with a positive intergenerational transmission of education and the fact that children to smokers are more likely to get respiratory problems (e.g., Cook and Strachan 1999), we would indeed expect these results.

  20. While using the 5th percentile to define “low physical capacity,” “weak handgrip strength,” “short,” “low visual acuity,” and “low hearing acuity” is somewhat arbitrary, we have experimented with using the 25th percentile, and this did not change our conclusions. Moreover, since the correlations between different health measures are not too strong, results are quite similar when including them one by one rather than simultaneously. See Appendix Tables 11 and 12 for these results.

  21. We can also account separately for either cognitive or noncognitive ability. These two are positively correlated, and controlling for either of them renders the effect of health small and insignificant.

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Acknowledgments

Research grants from the Centre for Economic Demography and from the Jan Wallander and Tom Hedelius Foundation are gratefully acknowledged. We thank two anonymous referees for their help and guidance.

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Correspondence to Anton Nilsson.

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This study was funded by the Centre for Economic Demography and the Jan Wallander and Tom Hedelius Foundation.

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The authors declare that they have no conflict of interest.

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Responsible editor: Erdal Tekin

Appendix

Appendix

Table 7 Descriptive statistics (population sample and DZ twins)
Table 8 Effect at different margins of schooling
Table 9 Effect of different margins of health
Table 10 Interactions with socioeconomic background
Table 11 Using the 25th percentiles to define poor health (low physical capacity, low handgrip strength, short height, poor visual acuity, and poor hearing acuity)
Table 12 Including health variables one by one

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Lundborg, P., Nilsson, A. & Rooth, DO. The health-schooling relationship: evidence from Swedish twins. J Popul Econ 29, 1191–1215 (2016). https://doi.org/10.1007/s00148-016-0598-8

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