Advertisement

Acta Endoscopica

, Volume 32, Issue 2, pp 157–166 | Cite as

Infections de l'intestin grêle

  • C. Cuvelier
Article

Résumé

Les infections intestinales grêles sont, avec les infections coliques, les maladies les plus fréquentes au monde et particulièrement agressives, spécialement chez les jeunes enfants, où elles peuvent causer un nombre élevé de décès dus aux diarrhées. Elles sont causées par des agents pathogènes les plus divers et leurs aspects pathologiques et épidémiologiques comme leur incidence peuvent être différents d'une contrée à l'autre.

Dans cet article, nous traiterons des causes pes plus communes des infections grêles et décrirons leurs caractéristiques cliniques et leurs aspects histologiques. Les maladies infectieuses sont classées en deux catégories, aiguës et chroniques; le type d'infiltrat inflammatoire peut aider à poser un diagnostic correct.

Mots-clés

cellules M, MALT entérite aiguë entérite chronique infections de l'intestin grêle 

Infections of the small intestine

Summary

Small intestinal infections are, together with colonic infections, amongst the most frequent occurring diseases worldwide and they harass mankind. Especially among young children there are enormous numbers of deaths due to diarrhoeal disease. They can be caused by a great diversity of pathogens and their pathological and epidemiological features as well as their incidence can often differ in different areas of the world.

In this paper the most common causes of small intestinal infections are reviewed and some characteristics of their clinical and histological aspects are described. Moreover, the different infectious diseases are classified into acute and chronic as the recognition of the type of inflammatory infiltrate may be help ful to make a correct diagnosis.

Key-words

acute enteritis chronic enteritis MALT M cells small intestinal infections 

Preview

Unable to display preview. Download preview PDF.

Unable to display preview. Download preview PDF.

Références

  1. 1.
    FASANO. — Bacterial infections: small intestine and colon.Curr. Opin. Gastroenterol., 2001,17, 4–9.PubMedCrossRefGoogle Scholar
  2. 2.
    CIARLET M., ESTES M.K. —Rotavirus and calicivirus infections of the gastrointestinal tract.Curr. Opin. Gastroenterol., 2001,17, 10–16.PubMedCrossRefGoogle Scholar
  3. 3.
    HUSTON C.D., PETRI Jr W.A. — Emerging and reemerging intestinal protozoa.Curr. Opin. Gastroenterol., 2001,17, 17–23.PubMedCrossRefGoogle Scholar
  4. 4.
    MÖNKEMÜLLER K.E., WILCOX C.M. — Gastrointestinal infections in children.Curr. Opin. Gastroenterol., 2001,17, 35–39.PubMedCrossRefGoogle Scholar
  5. 5.
    VAN SPREEUWEL J.P., DUURSMA G.C., MEIJER C.J.L.M., BAX R., ROSEKRANS P.C.M., LINDEMAN J. —Campylobacter colitis: histological, immunohistochemical and ultrastructural findings.Gut, 1985,26, 945–951.PubMedCrossRefGoogle Scholar
  6. 6.
    VAN TRAPPEN G., AGG H.O., PONETTE E., GEBOES K., BERTRAND Ph. —Yersinia enteritis and enterocolitis: gastroenterological aspects.Gastroenterol., 1977,72, 220–227.Google Scholar
  7. 7.
    CUVELIER C., DEMETTER P., MIELANTS H., VEYS E.M., DE VOS M. — The interpretation of ileal biopsies: morphological features in normal and diseased mucosa.Histopathology, 2001,38, 1–12.PubMedCrossRefGoogle Scholar
  8. 8.
    OBERT G., PEIFFER I., SERVIN A.L. — Rotavirus-induced structural and functional alterations in tight junctions of polarized intestinal Caco-2 cell monolayers.J. Virol., 2000,74, 4645–4651.PubMedCrossRefGoogle Scholar
  9. 9.
    MERLIN T.L., GIBSON D.W., CONNOR D.H. — Infectious and parasitic diseases, pp. 337–452.In: E. Rubin, H. Farber, Pathology, 2nd ed., 1994.Google Scholar
  10. 10.
    IKEDA K., KUMASHIRO R., KIFUNE T. — Nine cases of acute gastric anisakiasis.Gastrointest. Endosc., 1989,35, 304–308.PubMedCrossRefGoogle Scholar

Copyright information

© Springer-Verlag 2002

Authors and Affiliations

  • C. Cuvelier
    • 1
  1. 1.Department of Pathology, UZ GhentGhent UniversityGhent(Belgium)

Personalised recommendations