Anterograde and retrograde degeneration of corticospinal fibers after striatocapsular infarction
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A 40-year-old woman presented with sudden right-sided weakness and dysarthria. She had hypertension and dyslipidemia, without history of stroke. She was a non-smoker and overweight (body mass index 24.3 kg/m2). Her brother had a stroke at the age of 47. A neurological examination showed facial palsy, mild decrease in sensation, and hemiparesis on the right side. She was normal otherwise, including normal cortical function test and cranial nerve examination. She visited emergency room 30 min after symptom onset and received intravenous thrombolysis with recombinant tissue plasminogen activator after brain CT. Laboratory examinations, including 24-h Holter electrocardiography, were unremarkable. Initial magnetic resonance imaging (MRI) revealed an acute ischemic lesion involving the left caudate, putamen, and internal capsule, suggesting left striatocapsular infarction (Fig. 1a). Physical therapy was administered during admission, but she did not receive physical therapy after...
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The authors declare that they have no conflict of interest.
A single-case report without containing personally identifiable information was an exemption from formal ethics review in the Korea University Medical Center Institutional Review Board. Instead, we obtained a patient consent.
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Not commissioned; externally peer reviewed.
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